(Boston) — Dr. David Levin, Professor of Molecular & Cell Biology at Boston University Henry M. Goldman School of Dental Medicine and Professor of Microbiology at Boston University School of Medicine discovered recently a novel, evolutionarily con…
TORONTO, March 2, 2011 — York University researchers have zeroed in on a genetic process that may allow ovarian cancer to resist chemotherapy.
Researchers in the university’s Faculty of Science & Engineering studied a tiny strand of our geneti…
A lab headed by a Saint Louis University researcher has made a major breakthrough that could lead to a better molecular understanding of cancer. Results published today in the Journal Molecular Cell by Ali Shilatifard, Ph.D., and colleagues show for the first time how a protein known to be involved in the development of cancer functions in normal cells. The research shows how the protein “Bre1” plays a pivotal role in determining how the protein “Rad6” functions in modification of chromosomal DNA. Also participating in this research was the lab of Dr. Mark Johnston at Washington University School of Medicine.
In an embryo, certain genes must turn on to, for example, tell cells to develop into a limb. But just as importantly, the genes must then turn off, or go silent, to prevent abrnomral growth. How the genes do that gets some new light in research released out of North Carolina.