I am heartened that the tide seems to be turning. The Great Barrington Declaration is attracting thousands of scientists’ signatures each day.
The global response to COVID claims science for its foundation, and my aim in this series is to show that what is being done does not represent a scientific consensus, and is deeply variant from past public health practices. I don’t understand who is behind this, but I suspect that it is not mere incompetence or bureaucratic inertia; this suspicion is based on
- Fraudulence of chloroquine trials
- Suppression of scientific dissent
- Evidence that SARS-CoV-2 originated in a lab, and suppression of this evidence in the scientific literature and in the press
- Secrecy in planning the political response to COVID
- Neglect of all the ancillary harms from lockdown in deciding on a response. (This warning published last March in the NYTimes by a senior epidemiologist from Yale probably could not be published in October.)
- Well-established, safe and effective treatments for COVID are being bypassed to hang the world’s future on the mirage of a vaccine, though vaccines are (1) far more expensive and (2) much harder to prove safe and effective [See #10 below]
- Public announcements and even the way the numbers are calculated are inciting widespread fear in the public. I think this fear is far more than is warranted, and I suspect that this is by design.
The method behind this madness remains elusive to me. But political journalists outside the established media are emphasizing the military connection. One investigative journalist whom I respect for her courage and her diligence is Whitney Webb. Here, she shows us that Operation Warp-speed is a military project much more than a public health project. It is plausible to me that COVID originated in a bioweapons research lab. And from the beginning, the US response was planned not by public health experts but by secret meetings of military leaders.
I hope you will explore these connections and come to your own conclusions. My more modest goal in this series is to establish that “science” cannot be invoked to justify the lockdowns, the masking, the secrecy, the closure of schools and churches and cultural institutions. Least of all can “science” justify censorship, because the process by which science reaches for truth depends on open debate from a diversity of perspectives.
6. “New cases of COVID are expanding now in a dangerous Second Wave”
We’re concerned not for the virus but for the suffering and death that it causes. In March and April, we were frightened by the rising numbers of COVID deaths. But in May, CDC stopped reporting daily deaths and switched to reporting daily cases.
Traditionally, “cases” are defined as people who become seriously ill. That was the definition for a short while. Then it was “people who test positive for the virus”. On May 19, CDC started adding people who tested positive for antibodies to the virus as “cases”. We’re told that there is a troubling increase in COVID cases lately. If people really were getting sick, this would be disturbing. But if it is an increase in perfectly healthy people testing positive for antibodies, it is a wholly good thing. It’s called “herd immunity”.
No test is infallible, and invariably there are people who test positive who don’t really have the virus. These are false positives. As the prevalence of COVID has dropped with summer weather and more of the population already exposed (herd immunity), the rates are so low in many urban areas that false positive tests are swamping the true positives, and we really can’t say anything about trends. This recent article concludes that the quality of available data is no longer a reliable basis for policy data decisions.
https://www.nytimes.com/2020/08/29/health/coronavirus-testing.html
The low death rates are, of course, a good thing. The problem is that the false positives are being reported without explanation as though they were meaningful data about prevalence of COVID.
COVID is no longer among the top 5 causes of death in America. Why is our government slanting the reports in ways that keep us scared? I don’t have an answer to this question. I know there is a great deal of money riding on vaccines, and that by any sane criterion, COVID vaccines are past their usefulness, even if we had reason to believe they were safe. But I don’t think this fully explains the fear campaign. I suggest that it’s my job and yours to keep asking questions.
7. “Dr Fauci and the CDC are guiding our response to COVID according to the same principles of epidemic management that have protected public health in the past.”
On the contrary, standard public health procedure is to quarantine the sick and protect the most vulnerable. Telling a whole country full of healthy people to stay at home is entirely new, unstudied, a sharp departure from previous practices.
Closing down manufacturers, offices, stores, churches, concert halls, theaters, even closing private homes to social and family guests—all this is a radical new experiment. There are no scientific studies to justify it, because it has never been done in the past.
Containment of the virus is feasible if it is begun very early, when the virus is geographically contained and the number of cases is small enough that every case can be accounted for. It’s then possible for severe isolation to halt the virus in its tracks. (This was the strategy pursued by China.) Once there are thousands of cases, it is feasible to slow the spread, but not to change the fact that eventually, everyone in the population will be exposed.
Dr Fauci was clearly aware of this, because when he made his March announcement, he was asking America to isolate only for a few weeks. His goal was explicitly to “flatten the curve”, meaning to make sure the disease didn’t spread so rapidly that hospital ICUs would be overwhelmed. At the beginning, he (quite reasonably) did not claim that the measures he prescribed to America would contain the virus, but only slow its spread.
It worked. Except in a few isolated regions, there was never a shortage of hospital beds. But six months later, we are still masking and social distancing, long past when the original justification for these measures has been forgotten.
8. “Asymptomatic carriers are an important vector of disease transmission, which must be isolated if we are to stop the spread of COVID”
The justification for separating healthy people from other healthy people is the idea that we never know who is really healthy. We know from past history that colds and flu become contagious a day or two before they have symptoms, though the viral load that they transmit is greatly increased once the virus has taken hold and they are coughing and sneezing.
Extending quarantine from the traditional application to people who are obviously sick to the general population is a huge innovation, imposing tens of trillions of dollars in lost productivity worldwide, as well as social and psychological hardship. Isolation kills. It could only have been justified by evidence that the virus could not be contained by the same methods that have been used for all previous epidemics. Where is the evidence that asymptomatic carriers are a critical link in the chain of transmission?
Dr Fauci got it right at first when he said, “In all the history of respiratory-born viruses of any type, asymptomatic transmission has never been the driver of outbreaks. The driver of outbreaks is always a symptomatic person.” [] Subsequently, there were anecdotal articles documenting particular cases in which asymptomatic transmission did occur [one, two, three]. How can we know if asymptomatic carriers are an important part of the dynamic spread of the disease? This paper is the only attempt I have found to study the question with a detailed mathematical model; but, in the end, it just calculates unknowns from unmeasurables, and reaches no conclusion. We are left with common sense, which says that patients with symptoms have much higher viral levels (that’s why they are sick). They are also coughing and aspirating more of the virus (that’s why the virus evolved to make us cough). When Maria van Kerkhove, speaking for the WHO, , she was reined in by those who control the narrative, and she the statement the next day.
9. “The lower death rates now compared to April are due to protective measures such as social distancing, mask-wearing, and limited travel.”
Why would we expect lower death rates? From measures intended to limit social contact and spread of the virus, we should expect lower infection rates. But that’s not happening; instead, we have higher case rates coupled with lower death rates. This can reasonably be explained by (1) changes in definition of what constitutes a “case” (see #6 above), (2) wider testing, (3) the virus evolving, as most viruses tend to do, toward higher infectivity and lower fatality, and (4) fall weather.
10. “With enough resources, pharmaceutical scientists can develop a vaccine in a matter of months, and provide reasonable assurance that it is safe.”
This is the most dangerous of all the fictions and, not incidentally, the one most closely related to $6 billion in NIH investments and tens of billions in projected corporate profits.
The subject of vaccines is highly polarizing. On the one hand, the mainstream press, especially the scientific press, has been hammering with singular purpose the message that vaccines are safe and effective and necessary not just for individual protection but for public health. On the other hand, there is about one third of the American public who distrust what they hear about vaccines, enough so that they will refuse a vaccine (if not coerced). So much has been written about vaccine safety that I would not presume to try to convince you one way or the other in a few paragraphs. I can tell you that my own attitude changed when I had a bad reaction four years ago to a pneumonia vaccine (PCV13), and learned that there is no corporate liability for vaccine injuries. An act of Congress in 1986 exempted vaccines from the standard testing for safety and efficacy that other medications must pass, and also indemnified vaccine companies from all liability for harm caused by either design or manufacture. In my opinion, this is a dangerous situation, as it removes all motivation for companies to make a safe product.
I’ll close this series by defending my claim above that, compared to treatments, vaccines are (1) far more expensive and (2) much harder to prove safe and effective.
- One reason that vaccines are more expensive for the public (and correspondingly more profitable for the industry) is that vaccines are for everyone, while treatments are only for less than 1% of the population that becomes sick enough to need them. There is a race to patent a vaccine, a race for billions of dollars in private profits that derive from spending public research funds, and the profit potential is distorting our public priorities. The best treatment we have is hydroxychloroquine, which is out of patent, has a 65-year safety record, and costs pennies per dose. FDA can only legally approve vaccines on a fast track basis if they find that no viable treatments are available. This is ample explanation for the campaign to discredit chloroquine and other effective treatments.
- Because a vaccine is given to 100 times as many people, it must be 100 times safer in order to impose the same health burden from side effects. COVID is only life-threatening for people who are old and/or disabled; so to establish the safety of a vaccine, clinical trials must include people who are old and/or disabled. The relevant question is: are people who receive the vaccine dying at a lower rate than people who received a placebo? But none of the trials are being designed to ask this question.
There is a reason why vaccines are tested over many years, and why “warp-speed” testing cannot tell us what we need to know. Though a vaccine is always designed with one particular pathogen in mind, the effects of vaccination—beneficial and detrimental—extend to the immune system generally. This is the complex subject of cross-immunity [ref, ref, ref, ref]. It is that live virus vaccines tend to confer cross immunity toward non-target viruses, while vaccines made from protein fragments tend to impair immunity to non-target infections. Only one of the candidate vaccines is derived from live, attenuated virus. The new class of RNA vaccines [Moderna] is entirely untested, and we have no idea what the long-term effects would be, but initial results give us pause.
If you are open to an honest and competent criticism of vaccine science and politics, I recommend Robert F. Kennedy’s web site.
The Bottom Line
The story that we are being told about an ultra-lethal virus that “jumped to humans” and the scientific community converging on a response proportional to the threat—this story is unraveling, as more and more doctors and public health professionals are adding their voices to a global movement to restore sanity and integrity in the pandemic response.
Discussion
65 reader comments
Imported threads are marked Archive. New comments are welcome and moderated for spam.
This is a test of the re-Captcha Ben installed to fight the spam building up on Josh's site
I thought at the time that the pandemic was drawing to a close as the winter season ended. I was wrong about that.
I was correct, however, that the vaccines were vastly less effective than the treatments that were already available. Now, of course, there are even more effective treatments. Side effects from the treatments are minimal, and confined to those few who are sick. Side effects of the vaccines, in contrast, are serious, including heart damage, nerve damage, and death. And the vaccine side effects are spread over a much larger population trying to prevent the disease, not just the much smaller number who get sick from it.
-JJM
I just re-read this blog and noticed something I had not thought about before.
"by any sane criterion, COVID vaccines are past their usefulness". Could you clarify that?
Recent Research Paper from John P.A. Ioannidis et al., suggesting that the Lockdowns had “…no significant benefits…”. A damning indictment of the ‘Lockdown mentality’ perhaps?
Assessing Mandatory Stay‐at‐Home and Business Closure Effects on the Spread of COVID‐19
https://onlinelibrary.wiley.com/doi/10.1111/eci.13484
Let's take a close look at the 'science' which the 'covid pandemic' depends upon. There are two studies that are the foundation of this entire narrative. Here they are:
https://wwwnc.cdc.gov/eid/article/26/6/20-0516_article?fbclid=IwAR0HYPDhZoUX1NDOZrad1Y7VKZiBRGoJysdQ4sE_DBMDoAmd_P6gqfFx2w4
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6988269/?fbclid=IwAR1lW3dTNDKx56gqBw-6Axp00XDH7MgclAHH7HWBjvW02S7iQA3pWrwLasc
Here are some key quotes from the first study:
"We designed 37 pairs of nested PCRs spanning the genome on the basis of the coronavirus reference sequence (GenBank accession no. NC045512). We extracted nucleic acid from isolates and amplified by using the 37 individual nested PCRs. We used positive PCR amplicons individually for subsequent Sanger sequencing and also pooled them for library preparation by using a ligation sequencing kit (Oxford Nanopore Technologies, https://nanoporetech.comExternal Link), subsequently for Oxford Nanopore MinION sequencing. We generated consensus nanopore sequences by using Minimap version 2.17 (https://github.comExternal Link) and Samtools version 1.9 (http://www.htslib.orgExternal Link). We generated consensus sequences by Sanger sequencing from both directions by using Sequencher version 5.4.6 (https://www.genecodes.comExternal Link), and further confirmed them by using consensus sequences generated from nanopore sequencing.
...we examined the capacity of SARS-CoV-2 to infect and replicate in several common primate and human cell lines, including human adenocarcinoma cells (A549), human liver cells (HUH7.0), and human embryonic kidney cells (HEK-293T), in addition to Vero E6 and Vero CCL81 cells." (those vero cells are monkey kidney cells)
Each cell line was inoculated at high multiplicity of infection and examined 24h post-infection. No CPE was observed in any of the cell lines except in Vero cells, which grew to greater than 10 to the 7th power at 24 h post-infection. In contrast, HUH 7.0 and 293T showed only modest viral replication, and A549 cells were incompatible with SARS CoV-2 infection.”
And now an interpretation of that study by Dr. Tom Cowan:
"First, in the section titled “Whole Genome Sequencing,” we find that rather than having isolated the virus and sequencing the genome from end to end, that the CDC “designed 37 pairs of nested PCRs spanning the genome on the basis of the coronavirus reference sequence (GenBank accession no. NC045512).”
To me, this computer-generation step constitutes scientific fraud. Here is an equivalency: A group of researchers claim to have found a unicorn because they found a piece of a hoof, a hair from a tail, and a snippet of a horn. They then add that information into a computer and program it to re-create the unicorn, and they then claim this computer re-creation is the real unicorn. Of course, they had never actually seen a unicorn so could not possibly have examined its genetic makeup to compare their samples with the actual unicorn’s hair, hooves and horn.
The researchers claim they decided which is the real genome of SARS-CoV-2 by “consensus,” sort of like a vote. Again, different computer programs will come up with different versions of the imaginary “unicorn,” so they come together as a group and decide which is the real imaginary unicorn."
"The shocking thing about the above quote is that using their own methods, the virologists found that solutions containing SARS-CoV-2 — even in high amounts — were NOT, I repeat NOT, infective to any of the three human tissue cultures they tested.
These virologists, published by the CDC, performed a clear proof, on their terms, showing that the SARS-CoV- 2 virus is harmless to human beings."
Now some quotes from an analysis of the second study by an international team of medical professionals with extensive expertise in the subject matter:
"Peer review of the RTPCR test to detect SARS-CoV-2 reveals 10 major scientific flaws at the molecular and methodological level: consequences for false positive results."
This paper will show numerous serious flaws in the Corman-Drosten paper, the significance of which has led to worldwide misdiagnosis of infections attributed to SARS-CoV-2 and associated with the disease COVID-19. There are ten fatal problems with the Corman-Drosten paper which we will outline and explain in greater detail in the following sections.
The first and major issue is that the novel Coronavirus SARS-CoV-2 (in the publication named 2019-nCoV and in February 2020 named SARS-CoV-2 by an international consortium of virus experts) is based on in silico (theoretical) sequences, supplied by a laboratory in China [1], because at the time neither control material of infectious (“live”) or inactivated SARS-CoV-2 nor isolated genomic RNA of the virus was available to the authors. To date no validation has been performed by the authorship based on isolated SARS-CoV-2 viruses or full length RNA thereof. According to Corman et al.:
“We aimed to develop and deploy robust diagnostic methodology for use in public health laboratory settings without having virus material available.” [1]
The focus here should be placed upon the two stated aims: a) development and b) deployment of a diagnostic test for use in public health laboratory settings. These aims are not achievable without having any actual virus material available (e.g. for determining the infectious viral load). In any case, only a protocol with maximal accuracy can be the mandatory and primary goal in any scenario-outcome of this magnitude. Critical viral load determination is mandatory information, and it is in Christian Drosten’s group responsibility to perform these experiments and provide the crucial data.
Nevertheless these in silico sequences were used to develop a RT-PCR test methodology to identify the aforesaid virus. This model was based on the assumption that the novel virus is very similar to SARS-CoV from 2003 as both are beta-coronaviruses.
The PCR test was therefore designed using the genomic sequence of SARS-CoV as a control material for the Sarbeco component; we know this from our personal email-communication with [2] one of the co-authors of the Corman-Drosten paper. This method to model SARS-CoV-2 was described in the Corman-Drosten paper as follows:
“the establishment and validation of a diagnostic workflow for 2019-nCoV screening and specific confirmation, designed in absence of available virus isolates or original patient specimens. Design and validation were enabled by the close genetic relatedness to the 2003 SARS-CoV, and aided by the use of synthetic nucleic acid technology.”
The Reverse Transcription-Polymerase Chain Reaction (RT-PCR) is an important biomolecular technology to rapidly detect rare RNA fragments, which are known in advance. In the first step, RNA molecules present in the sample are reverse transcribed to yield cDNA. The cDNA is then amplified in the polymerase chain reaction using a specific primer pair and a thermostable DNA polymerase enzyme. The technology is highly sensitive and its detection limit is theoretically 1 molecule of cDNA. The specificity of the PCR is highly influenced by biomolecular design errors.
Reliable and accurate PCR-test protocols are normally designed using between 100 nM and 200 nM per primer [7]. In the Corman-Drosten paper, we observe unusually high and varying primer concentrations for several primers (table 1). For the RdRp_SARSr-F and RdRp_SARSr-R primer pairs, 600 nM and 800 nM are described, respectively. Similarly, for the N_Sarbeco_F and N_Sarbeco_R primer set, they advise 600 nM and 800 nM, respectively [1].
It should be clear that these concentrations are far too high to be optimal for specific amplifications of target genes. There exists no specified reason to use these extremely high concentrations of primers in this protocol. Rather, these concentrations lead to increased unspecific binding and PCR product amplification.
The design variations will inevitably lead to results that are not even SARS CoV-2 related.
The WHO-protocol (Figure 1), which directly derives from the Corman-Drosten paper, concludes that in order to confirm the presence of SARS-CoV-2, two control genes (the E-and the RdRp-genes) must be identified in the assay. It should be noted, that the RdPd-gene has one uncertain position (“wobbly”) in the forward-primer (R=G/A), two uncertain positions in the reverse-primer (R=G/A; S=G/C) and it has three uncertain positions in the RdRp-probe (W=A/T; R=G/A; M=A/C). So, two different forward primers, four different reverse primers, and eight distinct probes can be synthesized for the RdPd-gene. Together, there are 64 possible combinations of primers and probes!
As it stands, the N gene assay is regrettably neither proposed in the WHO-recommendation (Figure 1) as a mandatory and crucial third confirmatory step, nor is it emphasized in the Corman-Drosten paper as important optional reassurance “for a routine workflow” (Table 2).
Consequently, in nearly all test procedures worldwide, merely 2 primer matches were used instead of all three. This oversight renders the entire test-protocol useless with regards to delivering accurate test-results of real significance in an ongoing pandemic.
As it stands, the N gene assay is regrettably neither proposed in the WHO-recommendation (Figure 1) as a mandatory and crucial third confirmatory step, nor is it emphasized in the Corman-Drosten paper as important optional reassurance “for a routine workflow” (Table 2).
Consequently, in nearly all test procedures worldwide, merely 2 primer matches were used instead of all three. This oversight renders the entire test-protocol useless with regards to delivering accurate test-results..."
"RT-PCR is not recommended for primary diagnostics of infection. This is why the RT-PCR Test used in clinical routine for detection of COVID-19 is not indicated for COVID-19 diagnosis on a regulatory basis.
“Clinicians need to recognize the enhanced accuracy and speed of the molecular diagnostic techniques for the diagnosis of infections, but also to understand their limitations. Laboratory results should always be interpreted in the context of the clinical presentation of the patient..."
(consider the 'asymptomatic carrier' hype here: "RT-PCR is not recommended for primary diagnostics of infection...results should always be interpreted in the context of the clinical presentation of the patient..." In other words, no clinical presentations + misuse of even a VALID PCR test cannot be interpreted as a definitive positive diagnosis!)
"Kim et al. demonstrate a highly variable 3’ expression of subgenomic RNA in Sars-CoV-2 [23]. These RNAs are actively monitored as signatures for asymptomatic and non-infectious patients [10]. It is highly questionable to screen a population of asymptomatic people with qPCR primers that have 6 base pairs primer-dimer on the 3 prime end of a primer (Figure 3).
Apparently the WHO recommends these primers. We tested all the wobble derivatives from the Corman-Drosten paper with Thermofisher’s primer dimer web tool [11]. The RdRp forward primer has 6bp 3prime homology with Sarbeco E Reverse. At high primer concentrations this is enough to create inaccuracies.
These are severe design errors, since the test cannot discriminate between the whole virus and viral fragments. The test cannot be used as a diagnostic for SARS-viruses."
"Testing the primer pairs specified in the Corman-Drosten paper, we observed a difference of 10° C with respect to the annealing temperature Tm for primer pair1 (RdRp_SARSr_F and RdRp_SARSr_R). This is a very serious error and makes the protocol useless as a specific diagnostic tool."
"Additional testing demonstrated that only the primer pair designed to amplify the N-gene (N_Sarbeco_F and N_Sarbeco_R) reached the adequate standard to operate in a diagnostic test, since it has a sufficient GC-content and the Tm difference between the primers (N_Sarbeco_F and N_Sarbeco_R) is 1.85° C (below the crucial maximum of 2° C difference). Importantly, this is the gene which was neither tested in the virus samples (Table 2) nor emphasized as a confirmatory test. In addition to highly variable melting temperatures and degenerate sequences in these primers, there is another factor impacting specificity of the procedure: the dNTPs (0.4uM) are 2x higher than recommended for a highly specific amplification. There is additional magnesium sulphate added to the reaction as well. This procedure combined with a low annealing temperature can create non-specific amplifications. When additional magnesium is required for qPCR, specificity of the assay should be further scrutinized.
The design errors described here are so severe that it is highly unlikely that specific amplification of SARS-CoV-2 genetic material will occur using the protocol of the Corman-Drosten paper.
SUMMARY CATALOGUE OF ERRORS FOUND IN THE PAPER
The Corman-Drosten paper contains the following specific errors:
1. There exists no specified reason to use these extremely high concentrations of primers in this protocol. The described concentrations lead to increased nonspecific bindings and PCR product amplifications, making the test unsuitable as a specific diagnostic tool to identify the SARS-CoV-2 virus.
2. Six unspecified wobbly positions will introduce an enormous variability in the real world laboratory implementations of this test; the confusing nonspecific description in the Corman-Drosten paper is not suitable as a Standard Operational Protocol making the test unsuitable as a specific diagnostic tool to identify the SARS-CoV-2 virus.
3. The test cannot discriminate between the whole virus and viral fragments. Therefore, the test cannot be used as a diagnostic for intact (infectious) viruses, making the test unsuitable as a specific diagnostic tool to identify the SARS-CoV-2 virus and make inferences about the presence of an infection.
4. A difference of 10° C with respect to the annealing temperature Tm for primer pair1 (RdRp_SARSr_F and RdRp_SARSr_R) also makes the test unsuitable as a specific diagnostic tool to identify the SARS-CoV-2 virus.
5. A severe error is the omission of a Ct value at which a sample is considered positive and negative. This Ct value is also not found in follow-up submissions making the test unsuitable as a specific diagnostic tool to identify the SARS-CoV-2 virus.
6. The PCR products have not been validated at the molecular level. This fact makes the protocol useless as a specific diagnostic tool to identify the SARS-CoV-2 virus.
7. The PCR test contains neither a unique positive control to evaluate its specificity for SARS-CoV-2 nor a negative control to exclude the presence of other coronaviruses, making the test unsuitable as a specific diagnostic tool to identify the SARS-CoV-2 virus.
8. The test design in the Corman-Drosten paper is so vague and flawed that one can go in dozens of different directions; nothing is standardized and there is no SOP. This highly questions the scientific validity of the test and makes it unsuitable as a specific diagnostic tool to identify the SARS-CoV-2 virus.
9. Most likely, the Corman-Drosten paper was not peer-reviewed making the test unsuitable as a specific diagnostic tool to identify the SARS-CoV-2 virus.
10. We find severe conflicts of interest for at least four authors, in addition to the fact that two of the authors of the Corman-Drosten paper (Christian Drosten and Chantal Reusken) are members of the editorial board of Eurosurveillance. A conflict of interest was added on July 29 2020 (Olfert Landt is CEO of TIB-Molbiol; Marco Kaiser is senior researcher at GenExpress and serves as scientific advisor for TIB-Molbiol), that was not declared in the original version (and still is missing in the PubMed version); TIB-Molbiol is the company which was “the first” to produce PCR kits (Light Mix) based on the protocol published in the Corman-Drosten manuscript, and according to their own words, they distributed these PCR-test kits before the publication was even submitted [20]; further, Victor Corman & Christian Drosten failed to mention their second affiliation: the commercial test laboratory “Labor Berlin”. Both are responsible for the virus diagnostics there [21] and the company operates in the realm of real time PCR-testing." https://cormandrostenreview.com/report/?fbclid=IwAR2MM6P1bMsIYak5kbjfVxSKq_zjq9uS06vY8Kuett4LucIWCDkZtcitjHk
It boils down to this...the 'covid pandemic' narrative being hyped around the globe depends on an utterly fraudulent PCR test protocol that would not be able to accurately detect a positive covid virus infection EVEN IF the gene sequence had been conclusively established. But in fact, they are using a completely invalid test to 'detect' a virus that HAS NOT been conclusively gene sequenced, and regardless of that the virus identified has been shown to be non infectious in humans!
And now they are proceeding to mount a massive police state propaganda campaign, no doubt backed up by increasingly draconian mandatory vaccination statutes around the world, in order to foist their 'covid vaccines' on 7 billion people around the world, predicated on this fraudulent 'science'. And what do those mRNA vaccines all explicitly rely on? The gene sequence of the 'covid virus', which THEY DO NOT HAVE.
This line of thinking is not ageing particularly well. It comes across quite conspiratorial and over-wrought which is disappointing from a blogging voice that I've come to respect over the years. This line seems particularly ill-suited as a "false Covid narative":
6. “New cases of COVID are expanding now in a dangerous Second Wave”
11 million cases and counting in the US Josh. ... or are the people currently maxing out the intensive care wards across the nation just faking it?
Results from a October 29th study are promising.
"Hydroxychloroquine, nitazoxanide and ivermectin seem to be similarly effective for overall clinical outcomes in COVID-19 when used before seven days of symptoms, and overwhelmingly superior compared to untreated COVID-19 population, even for those outcomes not influenced by placebo effect, at least when combined with azithromycin, and vitamin C, D and zinc in the majority of the cases. Between these drugs, nitazoxanide demonstrated the strongest broad spectrum antiviral activity, plausibility to act as an anti-COVID agent, and safety profile, at least at the time of the choice of the drug for the AndroCoV Trial."
https://www.researchsquare.com/article/rs-98106/v1
I think the most telling study just came out in early September
Randomzied Controlled...they put newly asdmitted covid-19 patietns into 2 groups at the hospital..50 experimetnal 26 contols They gave everyone hydroxychloroquine and zinc. Then the gave the group of 50 an inital dose of around 100,000 IUs of vitamin d3 then 50,000 every other day...The result?
13 of the controls (50%) ended up in the ICU 2 of them died ..
The Vitamin D3 group of 50?? only 1 ended up in the ICU (2%) and none died. Conclusion? high dose d3 redfuced ICU admissions by 96%...Ta Da......
Check out my new podcast I did for the National Health federation it is just out.
youtube link>>> https://youtu.be/unScJt-Aliw
Alas, was it the D3, or was it D3 when combined with hydroxychloroquine and zinc? You can’t really say with such a study.
That's a good point. But there are a number of other studies out there that just look at d3 levels alone. For example one eregency room doctor measured all thne incoming covid-19 patient's D3 levels..and found not a single one admitted with a d3 blood level of more than 40 ng/ml... And no deaths in patients with a d3 blood level of 30 ng /ml or higher..I had seen a lot of studies like this where disease severity and death are all negatively correleated with d3 levels before this study popped up.So I had the prior background knowledge of Covid-19 and D3 so that is why I focused only on the D3 in this study...
Here are links to 13 studies. https://twitter.com/DrSubhasree/status/1305410804770992129
I would add that I have started being more diligent with D3, so am not disputing its value. Good to remember that for the previous millions of years before the advent of office spaces, we lived with a lot more sunlight. It’s no surprise that this compound is vital to health.
I’ll give a look see to your study , thnx.
Dr. Mitteldorf,
What is your best guess as to the real reasons behind these totally inane (actually insane) movements that you detail? I immediately saw them for what they were, not only with the misinformation, but also the absolute refusal to discuss time frames, when risk would be appropriate, what would a vaccine really change in people's minds (50% effective at best and with a coronavirus history of general ineffecacy), etc. When you don't get an answer you know you are dealing with emotion, hysteria, and general dishonesty. It's just not a good faith argument and it has never been.
If it's not bureaucratic creep or expert love, or money hungry and power hungry Gill Bates types with their connections to pharma/vaccine, what is your best guess? The stupidity and public reaction to this begs the question, truly. It has been an unbelievably heavy handed approach, to boot.
I thought it was extremely amusing that the search for super habitable planets include a temperature criteria of 5 degrees C warmer than Earth.
Here is a blurb about this criteria-
“Surface temperature of planets: The surface temperature of planets would strongly influence the formation of moisture, clouds, and humidity, all of which help determine the presence of a key life indicator: water. Planets with a mean surface temperature of about 5°C greater than Earth would be more suitable, as the slightly higher overall temperatures along with the additional moisture would be better for life. Life’s preference for warmth and moisture is evident on Earth too, as we see greater biodiversity in tropical rain forests as compared to colder, drier areas.“
You've misrepresented Dr. Maria Van Kerkhove's comments. While she did say that asymptomatic spread was not nearly as important as symptomatic spread, what she did say is that contact tracing and quarantine imposed on healthy individuals in contact with symptomatic individuals should be conducted.
In the absence of contact tracing and quarantining, a national policy for social distancing and limitations on social interaction that reduce physical contact is entirely merited and reasonable.
The Great Barrington Declaration is plain nonsense funded in part by the Kochs. It includes signatures from "Dr. Coconuts, PHD in Coconutology" and "Miss Wonderwoman, PHD in being bulletproof".
Now, the grifters behind the Great Barrington Declaration claim only .1% of the signatures are "fake", originating from "online trolls". Which means they allowed anyone with an internet connection to sign.
For another perspective, I suggest looking at "Science Based Medicine's" take on what these people are proposing
https://sciencebasedmedicine.org/great-barrington-declaration/
@Thomas,
You and the idiotic article you link too think that smearing other people's motives and pretending to be experts and part of the consensus is evidence that you are correct.
But to counter two of your outright lies
1. Many different groups have funded the Great Barrington Declaration from both sides of poltics)
2. Due to some assholes they now check signatures.. So 99.99% are real and it's over half a million in 3 weeks. Most people are not assholes it seems
If we really want to engage in conspiracy thinking, here is one hiding in plain sight.
The Great Barrington Declaration was sponsored by the American Institute for Economic Research (AIER), a libertarian think tank which receives a substantial part of its funding from its own investments, with holdings valued at US$284 million in a wide range of fossil fuel companies incl. Chevron and ExxonMobil, tobacco giant Philip Morris International, Microsoft, Alphabet Inc. and many other companies.[7][8] It has a balance sheet of US$37 million and in 2018 received a US$68,100 donation from the Koch Foundation. AIER describes itself as lobbying for a world "organized according to the principles of pure freedom – in which the role of government is sharply confined to the provision of public goods and individuals can flourish within a truly free market"[24] and as producing "independent, scientific, economic research to educate individuals, thereby advancing their personal interests and those of the nation". Its network of local "Bastiat Society" chapters partners with the Atlas Network, Ayn Rand Institute, Cato Institute, the Charles Koch Institute, and other Koch-funded think tanks. AIER statements and publications consistently portray the risks of climate change as minor and manageable, a form of climate change denial.
https://en.wikipedia.org/wiki/Great_Barrington_Declaration
It’s been awhile since I put much stock in Wiki, outside of botanical entries. They’ve consistently sided with the billionaires who’ve been pushing for the lockdown reaction to this cold virus, subsequent vaccine, etc. (i.e. Bill Gates).
Are they certain that AIER has been funding the work of these three scientists? And brought them to MA for this? “Sponsored” can mean a number of things.
"Sponsored" means they paid for it. Here's more info:
https://sciencebasedmedicine.org/great-barrington-declaration/
Besides, since when have libertarians been the "bad" guys? Unless its changed in recent years that I don't know about, libertarians have always been about liberty and allowing people the freedom to work it out on their own, making their own choices in life. They're the only people I know of that don't want to boss you around and make you do stuff you don't want to do. How in hell does this make libertarians the "bad" guys?
these people and groups are LINO'S. sinister groups with ulterior motives posing as liberty loving libertarians. you know, like antifa. classic fascists!!
i am no fan of the faux libertarian Koch bros. but after reading the Barrington declaration. Sign me up. the fact that fauci calls it nonsense is further proof that, that is the way to go.
Thank you, James - these are not organizations I would want to be associated with.
James, Thomas - are you really thinking of yourselves as “science-based” when pointing to sources that use word “denier”, a term of religious origin, indicating zelaous faith in whatever the mainstream academic circles deem the correct dogma for the time?
So, either you’re a “believer” in the dogma of catastrophic manmade global warming caused by CO2, or you are a “denier” and you should burn on stake with everyone who questions the Holy God of Science, right?
Settle down. "Denier" is simply a shorthand description for people who don't accept the obvious reality of anthropogenic global warming. No need to get dramatic about it.
Unfortunately, the word is used nowadays to shut down (or, like this case, shushing the opposing theory) all scientific discourse, be it climate, nutrition, medicine, etc, etc, etc. it’s a sophomore tool, an all too frequent trope.
I don’t think there is an opposing theory in Fox/Trump style of argumentation. There is just an over emphasis on plausible deniability and the assertion of conspiracy and persecution. There is a responsibility in science not only to be critical and examine alternative notions but to give due consideration to the weight of evidence. This also applies with COVID analysis.
There is no "alternative theory" plausibly explaining observed global warming. That's the problem.
I've never heard the term "denier" used in any other context. And it's frankly the most polite term I believe you could use to describe such people, though I'm open to suggestions.
There isn’t a single testable prediction from AGW that hasn’t been falsified. Be it Mann’s hockey stick or submersion of much of NYC or loss of the Maldives or collapse of global agriculture or increase in hurricanes or increase in tornados activity-all false by the use by date promoted.
Are you knowledgeable about paleo climate or the spectroscopy of carbon dioxide or the numerical modeling of chaotic natural systems?
If so please explain the basis of your certainty.