DALLAS — March 1, 2011 — Researchers at UT Southwestern Medical Center have identified a signaling pathway in the brain that’s sufficient to induce cellular leptin resistance, a problem that decreases the body’s ability to “hear” that it is full…
Tampa, Fla. (Dec. 23, 2010) — Two studies appearing in the current issue
of Cell Transplantation 19(10) discuss stem cells derived from adipose (fat) cells and their potential use in plastic surgery and tissue reconstruction. The studies are now…
From fad diets to exercise programs, Americans continue to fight the battle of the bulge. Now they’ll have help from recent Tel Aviv University research that has developed a new method to look at how fat cells — which produce the fat in our bodies …
Scientists are reporting new evidence that the fat tissue in those spare tires and lower belly pooches — far from being a dormant storage depot for surplus calories — is an active organ that sends chemical signals to other parts of the body, p…
Scientists are reporting new evidence that the fat tissue in those spare tires and lower belly pooches — far from being a dormant storage depot for surplus calories — is an active organ that sends chemical signals to other parts of the body, p…
The hormone leptin, primarily produced in fat cells, helps regulate food intake, metabolism and reproduction. It has also been shown to promote and sustain the body’s immune response by binding to T lymphocytes – the frontline cells that protect against infection.
The disease experimental autoimmune encephalomyelitis (EAE) in mice is currently used by researchers as a model of human multiple sclerosis (MS). Italian researchers now reports that just prior to developing the clinical symptoms of EAE, mice experience a significant burst of leptin which correlates with a reduction in food intake and weight loss. Furthermore, subjecting mice to acute starvation, which prevents the production of leptin, was found to delay the onset and reduce the severity of disease.